The University of Chicago Header Logo

Search Result Details

Back to Search Results
This page shows the details of why an item matched the keywords from your search.
Search Results
Wollmann, Robert
One or more keywords matched the following items that are connected to Wollmann, Robert
Item TypeName
Concept Receptors, Cholinergic
Academic Article A transgenic mouse model of the slow-channel syndrome.
Academic Article The human-severe combined immunodeficiency myasthenic mouse model: a new approach for the study of myasthenia gravis.
Academic Article Slow-channel transgenic mice: a model of postsynaptic organellar degeneration at the neuromuscular junction.
Academic Article Genetic manipulation of AChR responses suggests multiple causes of weakness in slow-channel syndrome.
Academic Article Striational autoantibodies in myasthenia gravis patients recognize I-band titin epitopes.
Academic Article Active calcium accumulation underlies severe weakness in a panel of mice with slow-channel syndrome.
Academic Article Focal caspase activation underlies the endplate myopathy in slow-channel syndrome.
Academic Article Rapsyn mutations in myasthenic syndrome due to impaired receptor clustering.
Academic Article Activation of apoptotic pathways at muscle fiber synapses is circumscribed and reversible in a slow-channel syndrome model.
Academic Article Mutations in MUSK causing congenital myasthenic syndrome impair MuSK-Dok-7 interaction.
Academic Article LG2 agrin mutation causing severe congenital myasthenic syndrome mimics functional characteristics of non-neural (z-) agrin.
Academic Article Refractoriness to a second episode of experimental myasthenia gravis. Correlation with AChR concentration and morphologic appearance of the postsynaptic membrane.
Academic Article Induction of the morphologic changes of both acute and chronic experimental myasthenia by monoclonal antibody directed against acetylcholine receptor.
Academic Article Acute severe animal model of anti-muscle-specific kinase myasthenia: combined postsynaptic and presynaptic changes.
Academic Article Desensitization of mutant acetylcholine receptors in transgenic mice reduces the amplitude of neuromuscular synaptic currents.
Search Criteria
  • Receptors Cholinergic